"Alternative Energy Pathways"

This is a phrase I used in my original write-up and one of my contentions that is probably among the most controversial and difficult to explain. I am going to try and explain what I understand about it, here. Please realize this is only a partial understanding from a limited set of knowledge.

With the initial treatment that creates the iatrogenic condition, the pituitary takes over regulation of electrolytes from the hypothalamus. It does this through hormonal control. This is a fundamental change in physiology. From there, various stages are entered over decades as a battle wages between the pituitary and candidiasis. The original article discussed how the pituitary was basically put into "overdrive." These changes impact a multitude of things, some immediately, some over time.

I am unclear on when this particular part changes, but I think it was with the initial change. What I am talking about is a change in how ATP is generated. The article stated, and I remember this distinctly, the condition is "all about ATP." Every phase, every change, is ATP related. Among the initial changes are two very significant ones. First, somehow through charges on blood cells, the circulatory system becomes inhospitable to the candidiasis. It is then forced into the surrounding cells. The modulation of the immune system that turns off the cellular defenses to candidiasis allows it to persist inside of cells. Once inside the cells, it produces ATP by reversing the sodium potassium pump. This obviously has issues, in that it treats the cell like a battery and eventually shrinks it and uses all the energy. This leaves the cells in an apoptotic state, essentially wrapping the candidiasis in a cellular prison (until later when sugars reach the cell and activate the candidiasis). ALL of that is not supported by currently available science. It is a travesty that the research has been redacted.

However, the article discussed something else that I have never done a good job explaining, and I am sure I won't do a great job of it today, 28 years after reading the article. The article talked about how due to pH changes, during most of the phases, insulin did not act normally. Somehow, the pH shift causes insulin to be less effective, but another energy pathway took over. This alternative pathway is a type of hemolysis. I believe this is related to the phagocytic process I have mentioned elsewhere.

I will attempt to explain what I can recall of the phagocytic process changes and it's effects. As the immune system is modulated by the initial treatment and the pituitary goes into overdrive, two things happen to the immune system. The first is the turning off of the some part that would normally defend the cells and recognize the invading candidiasis. The second modulation is some type of compensatory mechanism. Basically, because one part of the immune system is turned off, another part is turned UP. This part has to do with how dead necrotic cells are treated. Essentially, these cells are rapidly consumed by something. I'm not sure if the consumer is a white blood cell. It should be, but the article also talked about how the charges on the blood cells are changed and one type of cell can be mistaken for another type and/or age of cells due to the change in charge AND size of the cells.

However, this change has another dramatic effect. Whatever this change is, it also recognizes glycosylated cells as "prey" and consumes them. This process somehow also generates ATP. Over time, this becomes the dominant method of ATP generation. So, insulin does not work well, but blood sugar levels are maintained at normal levels because the red blood cells are consumed. As the article said, the patient has diabetes with normal blood sugar levels. Let's not go into the how, because I cannot explain the specifics. Instead, let's examine what you might expect to see as a result.

You might see anemia - I did test as anemic one time but that is it, and it was long ago. So, somehow my body has been producing enough blood cells to make up for the lost cells. You might see an irregular distribution of cells sizes indicating too many immature cells. However, this is accounted for by the changes in pH and the charges on the cells resulting in a swelling of the cells. The article was quite clear that blood counts would appear normal. A blood gas would/should show irregularities, but those are almost never done as they must be done by a physician. I've had one that I know of, and it was abnormal (overly basic, many years ago).

The other thing you might expect are high BUN levels. If red blood cells are being broken down at an abnormal rate, high BUN levels would be a primary expected finding. This is actually the most common abnormal result I have had over the years, often being in the 25-32 range.

Now, here one would argue that this is similar to PNH and should show up with hemoglobinuria. However, this is yet another insidious factor of the disease that prevents many expected warning signs from showing up in the urine. Here, I will readily lose every clinician because what I am saying is not present in any other condition and thus they have absolutely no frame of reference, as if someone simply deleted a chapter from their training. The suction effect of the heart, which is the primary cause of the onset of everything, happening at the very beginning, along with the reduction in blood volume causes the kidneys to stop functioning in their normal capacity. However, the suction somehow allows for filling of the bladder by filtering fluids from the overflow/leakage of the circulatory system. This process means the bladder wall acts as a filter preventing things like blood cells and excessive proteins from ever showing up in the urinalysis. I realize how absurd this sounds. I know how physiology works and what I am suggesting runs counter to everything that is taught about how physiology works. However, if you assume it to be true and work backwards and forwards to the logical conclusions, you will see that it would explain a lot. My kidney pain presented in early 2022. Upon drinking any amount of water, I would suffer tremendous back pain that would slowly subside. This was after the initial polyuria phase where I lost about 35 pounds via dark urine in 2 months. I'm fairly sure I documented it in my lengthy initial webpage.

The other thing that there really is no way to hide is the effect on the bones. The overproduction of red blood cells puts a strain on bone marrow. This is multiplied by the malnutrition caused by the other impacts of the overall condition on nutrient absorption due to changes in circulation to the gut. I have notable osteopenia, and have broken ribs with just my body weight resting on a hard surface as I went over an obstacle wall in a race.

The article discussed how, eventually, the marrow is unable to create red blood cells and can only produce the white blood cells which then attack other tissues in order to generate ATP.

So, I am sure I have some of that wrong and I know all of it is unconventional. However, the overall condition is not naturally occurring, and thus I believe it is reasonable to expect unconventional science to be part of the explanation.

On that note, I did have a Comprehensive blood panel performed yesterday and my Alkaline phosphatase levels are abnormally low while my BUN and BUN/Creatinine levels are high. Low alkaline phosphatase levels can indicate liver disfunction and/or bone marrow exhaustion. I think both are likely.

I want to add one thing to this analysis. The original experiments that I read about were conducted in the early 20th century. I can date them by the disease that caused the condition they were treating the patients for, the quality of the photos, the fact they had ice cream in containers, the use of a shot of adrenalin, and more. The science behind much of the explanation of the condition was not discovered until the 1930s and later (eg. the Kreb's cycle was discovered in 1937). So, those early 20th century treatments were not the only round of these experiments. The article mentioned the condition had been looked into for the super soldier program. I believe that inquiry and the poor outcome is why the science has been redacted. However, I am left wondering if treatments for the condition could have been developed that would prolong the life of someone with the condition. I cannot think of a reason why someone that was carefully monitored and aware of their condition could not be given a treatment regimen that would dramatically prolong the life of a subject with the condition. I managed to make it almost 28 years, and I know I did things that served to both prolong my and shorten my life expectancy.